Moffitt · Development and psychopathology 2002 · prospective longitudinal cohort study · n=?

Males on the life-course-persistent and adolescence-limited antisocial pathways: follow-up at age 26 years.

Level 3 - non-randomized controlled study

Prospective longitudinal cohort study (Dunedin Study follow-up to age 26).

PubMed 11893092 · doi:10.1017/s0954579402001104 · record verified 2026-08-26

What was done

This study evaluated adult outcomes at age 26 among males from the Dunedin longitudinal study categorized by the developmental timing of antisocial behavior: childhood-onset versus adolescent-onset delinquents (who were indistinguishable in adolescent offending), as well as a third group with childhood aggression but low adolescent delinquency. Assessed adult outcomes included personality traits, psychopathology, substance dependence, socioeconomic functioning, and criminal offenses.

What was found

The abstract reports no numerical data, proportions, or effect sizes. Qualitatively, at age 26: - Childhood-onset delinquents showed the highest elevations in psychopathic personality traits, mental health problems, substance dependence, number of children, financial and occupational difficulties, and drug-related and violent crime (including violence against women and children). - Adolescent-onset delinquents were less extreme but remained elevated on impulsive traits, mental health disorders, substance dependence, financial problems, and property crime. - Males with childhood aggression but low adolescent delinquency emerged as low-level chronic offenders who experienced anxiety, depression, social isolation, and occupational problems.

Why it matters

This study provides prospective evidence supporting developmental taxonomies of antisocial behavior into mid-twenties adulthood, demonstrating that childhood-onset delinquency leads to severe violence and comprehensive maladjustment, whereas adolescent-onset antisocial behavior does not fully remit by age 26.

Limits

The abstract does not report sample sizes (n), attrition rates, effect sizes, or test statistics. Findings are limited to males from a single birth cohort (Dunedin, New Zealand), precluding conclusions about females or broader populations without external replication. As an observational cohort study, causal mechanisms cannot be definitively isolated.

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