Pisani · Archives of physiology and biochemistry 2008 · meta-analysis of observational studies · n=?

Hyper-insulinaemia and cancer, meta-analyses of epidemiological studies.

Cited 340 times in the scientific literature.

Level 3 - non-randomized controlled study

Meta-analysis of non-randomized observational/cohort studies

PubMed 18465360 · doi:10.1080/13813450801954451 · record verified 2026-08-28

What was done

Authors searched PubMed for epidemiological studies (primarily prospective cohort studies using baseline or pre-diagnostic blood samples) investigating the relationship between markers of hyperinsulinemia and glycemia (insulin, C-peptide, glucose, HbA1c) and the risk of colon, rectum, pancreas, breast, and endometrial cancers. Multivariate-adjusted relative risks (RRs) and 95% confidence intervals were pooled in meta-analyses.

What was found

The meta-analyses identified excess risks of colorectal and pancreatic cancers associated with higher levels of circulating C-peptide/insulin and glycemic markers. For endometrial cancer (based on four studies), significant heterogeneity was observed, resulting in a summary RR compatible with no association. For breast cancer, while overall risk was higher in upper categories of C-peptide/insulin, this excess was driven entirely by retrospective studies. Specific numerical summary effect estimates and confidence intervals were not reported in the abstract.

Why it matters

It provides pooled epidemiological evidence that pre-diagnostic hyperinsulinemia and elevated glucose levels are linked to colorectal and pancreatic cancer development, helping clarify the metabolic mechanisms underlying obesity-related cancer risk.

Limits

The abstract reports no numerical risk estimates or total participant/study counts. The findings rely largely on single-point baseline blood measurements collected years before cancer diagnosis. For breast cancer, the association was limited to retrospective designs, introducing potential recall or reverse-causality biases, and significant heterogeneity was present across endometrial cancer studies.

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