Nestler · The Journal of clinical endocrinology and metabolism 1991 · Uncontrolled before-after interventional trial · n=6

A direct effect of hyperinsulinemia on serum sex hormone-binding globulin levels in obese women with the polycystic ovary syndrome.

Cited 825 times in the scientific literature.

Level 4 - case-series / case-control

Uncontrolled before-after interventional study in a small clinical series

PubMed 1898744 · doi:10.1210/jcem-72-1-83 · record verified 2026-08-29

What was done

Six obese women with polycystic ovary syndrome (PCOS) underwent ovarian steroid suppression with the GnRH agonist leuprolide depot (7.5 mg im on days -56, -28, and 0). While maintaining leuprolide, participants received oral diazoxide (300 mg/day) for 10 days to inhibit pancreatic insulin secretion. Serum sex steroids, sex hormone-binding globulin (SHBG), and insulin response to a 100-g oral glucose tolerance test (OGTT) were measured before and after diazoxide.

What was found

Leuprolide alone reduced total testosterone (1.72 ± 0.29 to 0.32 ± 0.09 nmol/L), non-SHBG-bound testosterone (104 ± 16 to 19 ± 5 pmol/L), androstenedione (7.25 ± 1.65 to 2.78 ± 0.94 nmol/L), estrone (371 ± 71 to 156 ± 29 pmol/L), estradiol (235 ± 26 to 90 ± 24 pmol/L), and progesterone (0.28 ± 0.12 to 0.08 ± 0.02 nmol/L), without altering SHBG (18.8 ± 2.8 to 17.8 ± 2.6 nmol/L). Subsequent diazoxide administration reduced OGTT insulin area under the curve from 262 ± 55 to 102 ± 33 nmol/min·L (P < 0.05). During diazoxide treatment, sex steroid levels remained suppressed and unchanged, but serum SHBG rose by 32% from 17.8 ± 2.6 to 23.5 ± 2.0 nmol/L (P < 0.003), and non-SHBG-bound testosterone dropped by 43% from 19 ± 5 to 11 ± 4 pmol/L (P = 0.05).

Why it matters

This study provides experimental evidence that hyperinsulinemia directly lowers circulating SHBG levels in obese women with PCOS independently of ovarian sex steroids.

Limits

The study is limited by an extremely small sample size (n = 6) and the absence of a parallel control group. Diazoxide was administered for only 10 days and caused deterioration of glucose tolerance, serving as an experimental probe rather than a practical therapeutic intervention.

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