Lane · Psychosomatic medicine 1990 · placebo-controlled clinical trial · n=25

Caffeine effects on cardiovascular and neuroendocrine responses to acute psychosocial stress and their relationship to level of habitual caffeine consumption.

Cited 145 times in the scientific literature.

Level 2 - randomized trial

Placebo-controlled clinical trial testing acute physiological responses in healthy volunteers

PubMed 2195579 · doi:10.1097/00006842-199005000-00006 · record verified 2026-08-31

What was done

Researchers evaluated the physiological effects of acute caffeine administration (3.5 mg/kg) compared to placebo in 25 healthy male volunteers categorized as habitual or light caffeine consumers. Cardiovascular and neuroendocrine markers—specifically blood pressure, plasma norepinephrine, plasma epinephrine, and cortisol—were measured under resting conditions pre- and post-substance administration, during an acute laboratory psychosocial stress task, and during a post-stress recovery period.

What was found

Caffeine significantly increased resting blood pressure and resting plasma norepinephrine, which produced additive elevations when combined with the stress task. Furthermore, caffeine potentiated stress-induced surges in plasma epinephrine and cortisol, more than doubling the magnitude of these hormonal responses compared to the placebo condition. These effects occurred in both light and habitual caffeine users without significant differences in response magnitude based on habitual intake levels.

Why it matters

This study demonstrates that caffeine can amplify both cardiovascular and endocrine responses to acute mental stress without evident tolerance developing from regular caffeine consumption.

Limits

The sample size was small (n = 25) and restricted entirely to healthy males, limiting generalizability to females and broader populations. The abstract reports qualitative directions and proportional changes (e.g., more than doubling) but lacks exact numerical values, confidence intervals, and details on randomization methods or long-term clinical endpoints.

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