Makimura · The Journal of clinical endocrinology and metabolism 2012 · randomized double-blind placebo-controlled trial · n=60

Metabolic effects of a growth hormone-releasing factor in obese subjects with reduced growth hormone secretion: a randomized controlled trial.

Cited 49 times in the scientific literature.

Level 2 - randomized trial

Individual randomized double-blind placebo-controlled trial.

PubMed 23015655 · doi:10.1210/jc.2012-2794 · record verified 2026-08-27

What was done

Sixty abdominally obese subjects with reduced growth hormone secretion were enrolled in a randomized, double-blind, placebo-controlled trial. Participants received either tesamorelin (a GHRH(1-44) analog, 2 mg daily) or placebo for 12 months. Visceral adipose tissue (VAT) and subcutaneous adipose tissue (SAT) were assessed by abdominal computed tomography scan, and carotid intima-media thickness (cIMT) was measured with ultrasound. Treatment effects were evaluated using longitudinal linear mixed-effects modeling.

What was found

Compared with placebo, tesamorelin significantly reduced VAT (-16 ± 9 vs 19 ± 9 cm2; treatment effect: -35 cm2, 95% CI -58 to -12, P = 0.003), cIMT (-0.03 ± 0.01 vs 0.01 ± 0.01 mm; treatment effect: -0.04 mm, 95% CI -0.07 to -0.01, P = 0.02), log C-reactive protein (-0.17 ± 0.04 vs -0.03 ± 0.05 mg/L; treatment effect: -0.15 mg/L, 95% CI -0.30 to -0.01, P = 0.04), and triglycerides (-26 ± 16 vs 12 ± 8 mg/dL; treatment effect: -37 mg/dL, 95% CI -67 to -7, P = 0.02). IGF-I increased in the tesamorelin group (treatment effect: 92 ug/L, 95% CI 52 to 132, P < 0.0001). No significant changes were observed in abdominal SAT (-6 ± 6 vs 3 ± 11 cm2, P = 0.40) or in fasting glucose, 2-hour glucose, and glycated hemoglobin. There were no serious adverse events or differences in adverse events between groups.

Why it matters

Augmenting endogenous growth hormone with tesamorelin selectively decreases visceral adiposity and improves cardiovascular risk markers without inducing adverse glycemic effects in obese patients with low growth hormone secretion.

Limits

The sample size was small (n = 60), limiting precision and power for secondary outcomes. The study was restricted to individuals with abdominal obesity and confirmed low growth hormone secretion, limiting generalizability to the broader obese population. Hard cardiovascular clinical outcomes were not evaluated.

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