Rogalski · Journal of cognitive neuroscience 2013 · Observational case-control and postmortem series · n=?

Youthful memory capacity in old brains: anatomic and genetic clues from the Northwestern SuperAging Project.

Cited 197 times in the scientific literature.

Level 4 - case-series / case-control

Cross-sectional case-control and postmortem series comparing selected elderly high-performers with controls.

PubMed 23198888 · doi:10.1162/jocn_a_00300 · record verified 2026-08-26

What was done

The Northwestern SuperAging Project evaluated community-dwelling individuals over age 80 with exceptional episodic memory performance (SuperAgers). Structural MRI was used to assess cortical thickness relative to age-matched cognitively average peers and younger healthy controls aged 50 to 60 years. Additional postmortem neuroanatomical assessments examined Alzheimer-type neuropathology and von Economo neuron density in the anterior cingulate cortex, alongside genetic testing for the apolipoprotein E (APOE) ε4 allele.

What was found

The abstract reports no numerical values, effect sizes, or test statistics. Qualitatively, SuperAgers showed higher cortical thickness than age-matched peers and greater anterior cingulate cortex thickness compared to healthy 50- to 60-year-old adults. A subset of SuperAgers had low densities of age-related Alzheimer pathology and high numbers of von Economo neurons in the anterior cingulate gyrus. The frequency of the APOE ε4 allele was lower in SuperAgers than in the general population.

Why it matters

The findings suggest potential neuroanatomical and genetic correlates—such as anterior cingulate cortex preservation and low APOE ε4 carriage—associated with resistance to age-related cognitive decline.

Limits

The abstract does not state the sample size (only noting a small cohort) or provide any quantitative measurements, confidence intervals, or p-values. Postmortem analyses were available only for an unspecified subset of cases, and the cross-sectional, observational design cannot prove causal resistance to neurodegeneration.

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