Xu · European journal of epidemiology 2017 · dose-response meta-analysis of prospective studies · n=11 studies (73,330 participants)

Alcohol consumption and dementia risk: a dose-response meta-analysis of prospective studies.

Cited 301 times in the scientific literature.

Level 3 - non-randomized controlled study

Systematic review and meta-analysis of prospective cohort studies

PubMed 28097521 · doi:10.1007/s10654-017-0225-3 · record verified 2026-08-29

What was done

Authors conducted a systematic search and random-effects dose-response meta-analysis of prospective observational studies evaluating the association between alcohol consumption and dementia risk. The analysis pooled data from 11 studies (73,330 participants; 4,586 all-cause dementia cases), 5 studies for Alzheimer's dementia (52,715 participants; 1,267 cases), and 4 studies for vascular dementia (49,535 participants; 542 cases).

What was found

A nonlinear association was found for all-cause dementia (p nonlinearity < 0.05). Risk reduction was confined to intake ≤12.5 g/day, with lowest risk at approximately 6 g/day (RR ≈ 0.9). All-cause dementia risk increased by approximately 10% when intake reached or exceeded 38 g/day (or 23 drinks/week). Wine showed prioritized associations over other types, and effects appeared stronger in individuals younger than 60 years old.

Why it matters

This meta-analysis identifies specific dose thresholds for dementia risk, demonstrating that any protective association is restricted to modest alcohol intake (≤12.5 g/day) and that heavier consumption (≥38 g/day) increases risk.

Limits

The analysis is restricted to observational prospective studies, preventing causal conclusions. The abstract does not report confidence intervals, exact risk estimates for dementia subtypes, or controls for the sick quitter bias or unmeasured lifestyle confounders.

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