Liu · Neurology 2017 · matched cohort study · n=386630

Vagotomy and Parkinson disease: A Swedish register-based matched-cohort study.

Cited 451 times in the scientific literature.

Level 3 - non-randomized controlled study

Nationwide non-randomized matched cohort study

PubMed 28446653 · doi:10.1212/WNL.0000000000003961 · record verified 2026-08-28

What was done

Nationwide Swedish register-based matched-cohort study of 9,430 vagotomized patients (3,445 truncal, 5,978 selective) identified between 1970 and 2010 and 377,200 general population reference individuals matched 40:1 by sex and birth year. Participants were followed up until Parkinson disease (PD) diagnosis, death, emigration, or December 31, 2010. Hazard ratios (HRs) and 95% confidence intervals (CIs) were estimated using Cox models adjusted for country of birth, chronic obstructive pulmonary disease, diabetes, vascular diseases, rheumatologic disease, osteoarthritis, and comorbidity index.

What was found

Over 7.3 million person-years of follow-up, 4,930 incident PD cases were identified. Incidence of PD per 100,000 person-years was 61.8 in vagotomized patients (80.4 truncal, 55.1 selective) and 67.5 in reference individuals. Overall vagotomy was not associated with PD risk (HR 0.96, 95% CI 0.78-1.17). Truncal vagotomy showed a trend toward reduced risk (HR 0.78, 95% CI 0.55-1.09), driven by procedures performed at least 5 years prior to PD diagnosis (HR 0.59, 95% CI 0.37-0.93). Selective vagotomy was not associated with PD risk.

Why it matters

These findings support the hypothesis that Parkinson disease pathology may originate in the gastrointestinal tract and ascend to the central nervous system via the vagus nerve.

Limits

Observational study design relying on administrative registers, potential confounding by unmeasured factors such as smoking, and possible misclassification of vagotomy type or diagnostic timing.

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