Mehdipour · Aging 2020 · controlled animal experiment and human ex vivo serum analysis · n=?

Rejuvenation of three germ layers tissues by exchanging old blood plasma with saline-albumin.

Cited 117 times in the scientific literature.

Level 5 - mechanism / opinion, no new human data

Preclinical animal model combined with human ex vivo serum proteomic testing (Level 5)

PubMed 32474458 · doi:10.18632/aging.103418 · record verified 2026-08-30

What was done

Old mice underwent a single neutral age blood exchange (NBE), replacing half of their plasma with saline containing 5% albumin. Investigators evaluated muscle repair, liver adiposity and fibrosis, and hippocampal neurogenesis. Comparative proteomic analysis was conducted on mouse NBE serum and human serum from therapeutic plasma exchange (TPE), alongside functional testing of human serum on progenitor cell proliferation.

What was found

The abstract does not provide specific numerical values, sample sizes, or effect sizes. Qualitatively, a single NBE matched or exceeded heterochronic blood exchange in improving muscle repair, decreasing liver adiposity and fibrosis, and increasing hippocampal neurogenesis in old mice. Proteomic analysis of mouse and human post-exchange serum showed a reset in systemic signaling factors, and a single human TPE reversed the age-associated inhibition of progenitor cell proliferation by old serum.

Why it matters

This study challenges the assumption that rejuvenation via blood exchange requires young systemic factors, demonstrating that diluting inhibitory factors in old blood is sufficient across tissues derived from all three germ layers.

Limits

In vivo functional and tissue rejuvenation outcomes were demonstrated only in mice. The human data are limited to ex vivo serum proteomic profiling and cell proliferation assays, without clinical outcome measures. Sample sizes, quantitative changes, and the duration of benefits were not reported in the abstract.

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