Ostrom · Free radical biology & medicine 2020 · randomized controlled trial · n=40

Aerobic exercise training partially reverses the impairment of Nrf2 activation in older humans.

Cited 33 times in the scientific literature.

Level 2 - randomized trial

Individual randomized controlled trial

PubMed 32866619 · doi:10.1016/j.freeradbiomed.2020.08.016 · record verified 2026-08-28

What was done

Young (aged 18–28 years, n = 21) and older (aged ≥60 years, n = 19) men and women were randomized to an 8-week aerobic exercise training program (3 days/week, 45 minutes/day) or a non-exercise control group. Before and after the 8-week intervention, participants completed an acute exercise challenge (30 minutes of cycling at 70% VO2peak). Peripheral blood mononuclear cells (PBMCs) were collected at 7 timepoints (pre-exercise, +10 m, +30 m, +1 h, +4 h, +8 h, +24 h) to measure Nrf2 nuclear localization, GCLC protein, and gene expression of NQO1, HO1, and GCLC.

What was found

Prior to training, older adults had significantly higher basal Nrf2 levels (p = 0.004) and a blunted acute Nrf2 signaling response compared to young adults (p = 0.05). Exercise training decreased basal Nrf2 expression compared to control (p = 0.032) and improved the acute Nrf2 signaling response in both young and older participants (p < 0.05). Restoration of the Nrf2 signaling response correlated with the change in basal Nrf2 (p = 0.039), an effect driven by older adults (p = 0.014). Lower basal nuclear Nrf2 was associated with greater acute exercise responses in Nrf2 and GCLC protein and NQO1 and GCLC mRNA.

Why it matters

This study provides randomized evidence that age-related blunting of Nrf2 antioxidant signaling is modifiable, showing that 8 weeks of aerobic exercise partially restores dynamic cellular redox responses in sedentary older adults.

Limits

The total sample size was small (40 participants across four sub-groups). Outcomes were surrogate molecular markers measured only in circulating PBMCs rather than skeletal muscle or other primary metabolic tissues, and clinical functional outcomes were not reported in the abstract.

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