Çolak · Thorax 2021 · Mendelian randomisation and prospective cohort study · n=116,335

Low vitamin D and risk of bacterial pneumonias: Mendelian randomisation studies in two population-based cohorts.

Cited 31 times in the scientific literature.

Level 3 - non-randomized controlled study

Prospective cohort study and Mendelian randomisation analysis

PubMed 33109689 · doi:10.1136/thoraxjnl-2020-215288 · record verified 2026-08-30

What was done

Researchers evaluated the association between plasma 25-hydroxyvitamin D levels and the risk of hospital-diagnosed bacterial pneumonia using observational and Mendelian randomisation designs. They studied 116,335 randomly chosen white Danish participants aged 20 to 100 years from the Copenhagen City Heart Study and the Copenhagen General Population Study followed from 1981 through 2018 (up to 38 years). Measured plasma 25-hydroxyvitamin D was available for 35,833 participants. Genetic analyses used instrumental variants around CYP2R1, DHCR7, GEMIN2, and HAL. Negative control outcomes (urinary tract infections, skin infections, sepsis, and gastroenteritis) were assessed to detect non-specific confounding.

What was found

During follow-up, 6,342 bacterial pneumonias occurred in observational analyses and 13,916 in genetic analyses. In multivariable-adjusted observational analyses, individuals with 25-hydroxyvitamin D <25 nmol/L had a higher risk of bacterial pneumonia compared to those with ≥25 nmol/L (HR 1.27, 95% CI: 1.16 to 1.40). In genetic analyses, each 10 nmol/L lower genetically predicted plasma 25-hydroxyvitamin D was associated with increased risk of bacterial pneumonia: Wald's ratio OR 1.12 (95% CI: 1.02 to 1.23), inverse-variance weighted OR 1.12 (95% CI: 1.04 to 1.20), MR-Egger OR 1.63 (95% CI: 0.96 to 2.78), and weighted median OR 1.15 (95% CI: 1.05 to 1.26). Associations were strongest for CYP2R1 variants. Neither observational nor genetic analyses showed associations with negative control infection outcomes.

Why it matters

These findings provide genetic evidence supporting a potential causal relationship between lifelong lower vitamin D concentrations and increased susceptibility to bacterial pneumonia, rather than the link being solely an artifact of reverse causation or lifestyle confounding.

Limits

The study population was entirely composed of white Danish individuals, limiting generalisability to other ancestral or geographic groups. Measured vitamin D levels were available in only a subset (35,833 of 116,335) of participants, the MR-Egger estimate had wide confidence intervals crossing the null, and outcomes were limited to hospital-diagnosed bacterial pneumonias, missing mild or outpatient-treated infections.

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