Lee · Journal of the American Heart Association 2021 · prospective cohort study · n=5580

Longitudinal Plasma Measures of Trimethylamine N-Oxide and Risk of Atherosclerotic Cardiovascular Disease Events in Community-Based Older Adults.

Cited 92 times in the scientific literature.

Level 3 - non-randomized controlled study

Prospective cohort study with serial biomarker measurements and longitudinal event follow-up.

PubMed 34398665 · doi:10.1161/JAHA.120.020646 · record verified 2026-08-30

What was done

Serial plasma trimethylamine N-oxide (TMAO) concentrations were measured at baseline and after 7 years using stable isotope dilution liquid chromatography-tandem mass spectrometry in community-dwelling older US adults. The study analyzed associations with centrally adjudicated incident ASCVD (n = 4131) and recurrent ASCVD (n = 1449). Multivariable Cox proportional hazards models adjusted for time-varying demographics, lifestyle factors, medical history, laboratory parameters, and diet, and tested for mediation and effect modification by estimated glomerular filtration rate (eGFR).

What was found

During follow-up, 1766 incident and 897 recurrent ASCVD events occurred. Comparing the highest to lowest TMAO quintile, elevated TMAO was associated with incident ASCVD (HR 1.21; 95% CI, 1.02–1.42; P-trend = 0.029), but adjusting for eGFR attenuated the association (HR 1.07; 95% CI, 0.90–1.27). eGFR significantly modified this relationship (P-interaction < 0.001): TMAO was associated with incident ASCVD in individuals with eGFR < 60 mL/min/1.73 m² (HR 1.56; 95% CI, 1.13–2.14; P-trend = 0.007), but not in those with eGFR ≥ 60 mL/min/1.73 m² (HR 1.03; 95% CI, 0.85–1.25; P-trend = 0.668). For recurrent ASCVD, higher TMAO was associated with increased risk (HR 1.25; 95% CI, 1.01–1.56; P-trend = 0.009) without significant eGFR modification.

Why it matters

The relationship between circulating TMAO and primary cardiovascular events in older adults is heavily modified or confounded by kidney function, whereas TMAO remains an independent marker for recurrent vascular events.

Limits

Observational cohort design cannot determine whether TMAO is a causal mediator or a non-causal marker of impaired renal clearance and subclinical disease. The study population was restricted to older US adults, limiting generalizability to younger age groups.

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