Chauhan · American journal of nephrology 2022 · retrospective cohort study · n=834

Prevalence and Outcomes Associated with Hyperuricemia in Hospitalized Patients with COVID-19.

Cited 21 times in the scientific literature.

Level 3 - non-randomized controlled study

Retrospective observational cohort study evaluating prognostic associations

PubMed 34883482 · doi:10.1159/000520355 · record verified 2026-08-27

What was done

Researchers evaluated 834 adult patients hospitalized for over 24 hours with COVID-19 in the Mount Sinai Health System who had at least one serum uric acid (UA) measurement. They analyzed the association between the initial serum UA level and the risk of acute kidney injury (AKI, KDIGO criteria), major adverse kidney events (MAKE: composite of all-cause in-hospital mortality, new dialysis, or ≥100% creatinine increase), in-hospital mortality, and inflammatory/cardiac biomarkers, adjusting for demographics, comorbidities, and laboratory values.

What was found

Median patient age was 66 years, 42% were female, and median baseline serum UA was 5.9 mg/dL (IQR 4.5–8.8). Overall, 60% developed AKI, 52% developed MAKE, and 32% died. In multivariable models, a doubling of serum UA was significantly associated with AKI (OR 2.8, 95% CI 1.9–4.1), MAKE (OR 2.5, 95% CI 1.7–3.5), and in-hospital mortality (OR 1.7, 95% CI 1.3–2.3). Higher UA levels were independently associated with higher procalcitonin (β = 0.6, SE 0.2) and troponin I (β = 1.2, SE 0.2), but showed no significant association with ferritin, C-reactive protein, or IL-6.

Why it matters

Elevated serum uric acid is an independent, dose-dependent prognostic marker for severe renal outcomes and death in hospitalized COVID-19 patients, identifying high-risk individuals early during admission.

Limits

Selection bias is a major concern because serum uric acid is not routinely measured in all COVID-19 admissions and was only available in a subset of patients. As an observational study, it cannot establish whether hyperuricemia is causally damaging to kidneys or merely a marker of severe catabolism, hypoxia, and reduced renal clearance. Post-discharge kidney recovery and long-term mortality were not assessed.

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