Manza · Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology 2024 · Double-blind, placebo-controlled crossover randomized trial · n=20

Brain connectivity changes to fast versus slow dopamine increases.

Cited 11 times in the scientific literature.

Level 2 - randomized trial

Individual double-blind, placebo-controlled randomized crossover trial

PubMed 38326458 · doi:10.1038/s41386-024-01803-8 · record verified 2026-08-29

What was done

Twenty healthy adults participated in a double-blind, placebo-controlled study comparing the effects of fast versus slow dopamine increases using methylphenidate (MP). Fast increases were induced via intravenous administration (0.5 mg/kg) and slow increases via oral administration (60 mg) during simultaneous [11C]raclopride PET and fMRI scans. The investigators evaluated minute-to-minute changes in global brain connectivity (GBC), compared whole-brain spatial patterns against normative maps of dopamine D1 receptor availability ([11C]SCH23390), and examined correlations with self-reported drug 'high' ratings.

What was found

Whole-brain connectivity patterns for fast versus slow dopamine increases were inversely correlated across space (rho = -0.54, p_spin < 0.001). Fast increases elicited greater GBC in the dorsal prefrontal cortex, insula, posterior thalamus, brainstem, caudate, and precuneus, whereas slow increases showed greater GBC in the ventral striatum, orbitofrontal cortex, and frontopolar cortex (p_FDR < 0.05). The fast > slow connectivity pattern correlated with normative D1 receptor availability (rho = 0.22, p_spin < 0.05). In addition, hippocampal GBC during fast dopamine increases negatively correlated with subjective 'high' ratings following intravenous administration (r(19) = -0.68, p_bonferroni = 0.015).

Why it matters

These findings demonstrate that the rate of dopamine elevation fundamentally alters large-scale brain network communication, providing a neurobiological mechanism for why rapid-onset stimulants produce distinct subjective reward and carry higher abuse liability.

Limits

The sample was small (n = 20) and composed solely of healthy adults, which limits generalizability to populations with substance use disorders. Dopamine D1 receptor distribution was estimated from normative external maps rather than measured directly in the participants.

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