Svart · Journal of nutrition and metabolism 2024 · randomized crossover trials · n=21

Three Weeks on a Ketogenic Diet Reduces Free Testosterone and Free Estradiol in Middle-Aged Obese Men and Women.

Cited 9 times in the scientific literature.

Level 2 - randomized trial

Two individual randomized crossover trials.

PubMed 39139216 · doi:10.1155/2024/9301369 · record verified 2026-08-30

What was done

Researchers integrated data from two randomized, controlled, crossover trials. Study I evaluated 10 healthy young men given a single oral weight-adjusted ketone ester or water control after an overnight fast, monitoring plasma beta-hydroxybutyrate and testosterone across 300 minutes. Study II assessed 11 middle-aged adults with obesity (5 men, 6 women) who completed 3 weeks of a ketogenic diet and 3 weeks of a standard diet in crossover fashion, measuring fasting sex hormones, sex hormone-binding globulin (SHBG), and calculated free hormone indices.

What was found

In Study I, acute ketone ester intake did not significantly alter testosterone relative to control (ketone ester dropped from 23.8 ± 2.4 to 22.3 ± 2.5 nmol/l; control rose from 20.9 ± 2.1 to 22.2 ± 1.9 nmol/l; p = 0.06). In Study II, 3 weeks of ketogenic diet did not alter total testosterone in men (20.2 ± 1.23 vs. 18.2 ± 1.23 nmol/l, p = 0.1) but lowered total testosterone in women (0.87 ± 0.06 vs. 1.1 ± 0.06 nmol/l, p < 0.0001). SHBG increased after the ketogenic diet in men (31.2 ± 2.6 vs. 25.0 ± 2.6 nmol/l, p < 0.0001) and women (26.5 ± 3.05 vs. 24.2 ± 3.05 nmol/l, p = 0.003). Free androgen index decreased in men (0.65 ± 0.05 vs. 0.74 ± 0.05, p = 0.04) and women (0.036 ± 0.006 vs. 0.05 ± 0.006, p = 0.0001). Free estradiol index also dropped in men (3.1 ± 0.8 vs. 4.8 ± 0.8, p = 0.0003) and women (1.2 ± 2.2 vs. 9.8 ± 2.2, p = 0.0001).

Why it matters

These findings suggest that sustained nutritional ketosis alters the hypothalamic-pituitary-gonadal axis by raising SHBG and decreasing bioavailable sex steroids in adults with obesity, while acute ketone ester ingestion shows minimal immediate hormonal effect.

Limits

Both cohorts were extremely small (n = 10 and n = 11). Study duration was limited to 3 weeks for the dietary intervention and 300 minutes for the acute challenge. The dietary cohort was restricted to middle-aged individuals with obesity, preventing direct generalization to lean athletic populations, and clinical endpoints related to the reduced free sex steroid levels were not measured.

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