Asami · Frontiers in cell and developmental biology 2025 · In vitro single-cell transcriptomic and functional perturbation study · n=?

Mouse and human embryonic genome activation initiate at the one-cell stage.

Cited 1 times in the scientific literature.

Level 5 - mechanism / opinion, no new human data

Bench in vitro and animal model transcriptomic study (non-clinical)

PubMed 40809689 · doi:10.3389/fcell.2025.1594995 · record verified 2026-08-26

What was done

Time-course single-cell RNA-sequencing (scRNA-seq) was conducted on one-cell mouse and healthy human embryos to profile early transcriptional activity after fertilization. In mouse one-cell embryos, functional disruption of candidate transcription factors (including MYC/c-Myc) was performed to test their requirement for embryonic development and immediate embryonic genome activation (iEGA).

What was found

In mouse embryos, iEGA initiated within 4 hours of fertilization primarily from the maternal genome, with paternal transcription appearing at approximately 10 hours. Healthy human one-cell embryos also showed significant low-magnitude transcriptional upregulation. In both species, early transcripts were canonically spliced. Inhibiting c-Myc in mouse embryos caused acute developmental arrest, disrupted iEGA, and caused the upregulation of hundreds of normally repressed genes. Specific numerical transcript counts, sample sizes, and effect sizes were not provided in the abstract.

Why it matters

These findings challenge the consensus that embryonic genome activation begins at the 2-cell stage in mice and the 4-to-8-cell stage in humans. Demonstrating transcription and active genome repression at the one-cell stage alters basic understanding of early mammalian development, epigenetic reprogramming, and embryonic arrest.

Limits

The abstract does not disclose sample sizes (n) for either mouse or human embryos, nor does it report exact statistical metrics, effect sizes, or confidence intervals. Functional inhibition experiments were conducted only in mouse embryos, leaving direct functional causality in human one-cell embryos unverified.

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