Smith · JAMA psychiatry 2025 · systematic review and meta-analysis of randomized controlled trials · n=127 randomized controlled trials (44,076 participants in meta-analysis; 163 trials / 54,962 participants in qualitative synthesis)

Antipsychotic Drugs and Dysregulated Glucose Homeostasis: A Systematic Review and Meta-Analysis.

Cited 12 times in the scientific literature.

Level 1 - systematic review of randomized trials

Systematic review and meta-analysis of randomized controlled trials

PubMed 40864439 · doi:10.1001/jamapsychiatry.2025.2240 · record verified 2026-08-27

What was done

The authors conducted a systematic review and random-effects meta-analysis of blinded randomized clinical trials (RCTs) searched across MEDLINE, Embase, PsychINFO, CINAHL, CENTRAL, and Web of Science through February 3, 2025. Studies evaluated patients with severe mental illness or healthy volunteers assigned to antipsychotics (APs) versus control (placebo or no intervention) and reported glucose metabolism parameters. Of 163 eligible RCTs (35,952 AP-treated and 19,010 placebo-treated patients), 127 RCTs (28,975 AP-treated and 15,101 placebo-treated patients) were quantitatively meta-analyzed. Primary outcomes included changes in fasting glucose, fasting insulin, and glycated hemoglobin (HbA1c); secondary outcomes included hyperglycemia incidence and insulin resistance. Subgroup analyses and metaregressions evaluated the effects of diagnosis, AP type, age, duration, dose, concomitant medication, and prior AP exposure.

What was found

Compared with placebo, AP treatment was associated with significant increases across all primary glucose markers: - Fasting glucose: mean difference (MD) 0.72 mg/dL (95% CI, 0.54–1.08; P < .001) - Fasting insulin: MD 1.94 µIU/mL (95% CI, 1.28–2.61; P < .001) - HbA1c: MD 0.04% (95% CI, 0.02%–0.05%; P < .001) - Hyperglycemia incidence: odds ratio 1.29 (95% CI, 1.04–1.59; P = .02) Findings were consistent in healthy volunteers. Metaregression and subgroup analyses showed dysglycemia risk was independent of exposure time, AP dose, AP type, diagnosis, age, concomitant medications, previous AP exposure, and weight gain propensity.

Why it matters

This study provides high-level evidence that antipsychotics directly impair glucose homeostasis independently of weight gain, drug type, or dosage. It establishes that metabolic monitoring is necessary for all patients receiving antipsychotics, rather than only those who gain weight.

Limits

The review was restricted to English-language publications. While statistically significant, the mean continuous changes across trials were small in absolute terms (e.g., 0.72 mg/dL for glucose, 0.04% for HbA1c), and the abstract does not report long-term rates of progression to clinical diabetes, specific treatment durations, or statistical heterogeneity metrics (such as I²).

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