HoldsUp

Mark Hyman, MD · 2025-06-18 · Andy Galpin, Mark Hyman (host)

The Real Reason You're Always Tired Has Nothing to Do With Sleep

32 claims checked against research: 2 contradicted 1 contradicted online 8 overstated 3 needing context 12 supported 6 unverified

2

Contradicted by research

1:07:55Andy Galpincontradictedvery low

Monozygotic twin research shows that individuals who do not lift weights exhibit lower muscle quality and lose power and speed more frequently as they age.

"We've done this research in our lab with twins, right? So monozygous twins, exact same DNA with different things. And when you look at the folks who don't lift weights, they don't tend to have as high muscle quality. They start to lose power and speed more frequently." (said at 1:07:55)

The speaker claims that monozygotic twin research from their lab demonstrates that individuals who do not lift weights exhibit lower muscle quality and lose power and speed more frequently. However, the published monozygotic twin study from this research group evaluated 30 years of discordant chronic endurance training (not resistance training/weightlifting) in a single pair of identical twins. In that study, the endurance-trained twin actually exhibited lower muscle size and strength as well as poorer muscle quality compared to the untrained twin, alongside a shift toward slow-twitch (MHC I) muscle fibers. No published monozygotic twin study demonstrating lower muscle quality or accelerated power/speed loss specifically resulting from a lack of weightlifting was identified.

1:26:00Andy Galpincontradictedlow

Analysis of the NHANES database revealed no correlation between muscle mass and strength training history, indicating that higher muscle mass in that survey population was driven by higher total body mass.

"We looked in that database, and what's interesting is there was no relationship between the amount of muscle mass people had and their strength training history really in that database. You know what that tells us? How did they get their muscle? If they had a lot of muscle, that had we had no bearing or understanding if they lifted weights or not, which means people in those national databases have generally accrued their muscle by just being bigger, larger people, ate more, bigger body mass comes along with more muscle." (said at 1:26:00)

The assertion that NHANES data show no relationship between muscle mass and strength training history is contradicted by published analyses of the NHANES database. A cross-sectional study of 4,872 U.S. adults from NHANES (1999–2004) demonstrated that muscle-strengthening frequency was significantly and positively associated with fat-free mass percentage in both men and women after adjusting for age, height, race-ethnicity, education, and caloric intake (PMID: 27031912). While it is well-established that absolute fat-free mass in general population surveys scales strongly with total body mass and fat mass (PMID: 27862528), strength training history does correlate significantly with body composition measures in NHANES.

1

Contradicted by web sources

0:57:37Mark Hyman (host)contradicted (web)

Larry Lewis ran five miles across the Golden Gate Bridge to work as a waiter at the age of 105.

"And this guy Larry Lewis was 105 years old. He lived in Marin in Northern California, and he would run across the Golden Gate Bridge every day, five miles to go to work as a waiter all day." (said at 0:57:37)

No published record matching the story of Larry Lewis running five miles across the Golden Gate Bridge to work as a waiter at age 105 was located; this does not prove the claim false.

contradicted by web sources

Historical reports and contemporary news archives record that Larry Lewis lived in San Francisco and did his daily morning run of over six miles through Golden Gate Park, not across the Golden Gate Bridge from Marin. Lewis then walked or jogged to his banquet waiter job at the St. Francis Hotel in downtown San Francisco before retiring at age 105.

oclc.orgnewspaperarchive.comwichita.edu

8

Overstated

0:13:10Andy Galpinoverstatedmoderate

Sleep restriction to five hours per night causes the brain to adapt by rapidly increasing the amount and speed of entry into deep sleep and REM cycles.

"and we're going to load you down to five hours of sleep per night. And then we're going to stay at five hours of sleep no matter what... You know what would happen to your deep sleep? It would rocket up. Why? Because your body would learn pattern recognition really quickly. "I don't have eight hours to mess around. I will get into deep, I will get into REM, I will get into these cycles hyper-fast because there's no wiggle room there."" (said at 0:13:10)

Restricting sleep to five hours per night does not cause the absolute amount of deep sleep (slow-wave sleep, SWS) to "rocket up," nor does it increase total REM sleep. Under chronic sleep restriction, homeostatic sleep pressure prioritizes slow-wave sleep such that its absolute duration is largely preserved relative to baseline, leading to an increased relative proportion (percentage) of SWS within the shortened sleep window. However, total REM sleep duration is typically reduced because REM sleep predominates in the later hours of a full night's sleep. Furthermore, faster entry into deeper stages is driven by homeostatic pressure (adenosine accumulation and Process S), not cognitive pattern recognition or biological learning.

0:17:01Andy Galpinoverstatedmoderate

Elevated hematocrit and hemoglobin in physically unfit, non-exercising individuals is a common clinical indicator of sleep apnea.

"Really good example: if you see somebody who doesn't exercise, they're unfit, but you see hematocrit and hemoglobin are really high, really good chance you've got some sort of apnea." (said at 0:17:01)

While obstructive sleep apnea (OSA) can induce mild nocturnal hypoxia leading to slight, measurable increases in hemoglobin and hematocrit, clinically significant erythrocytosis or 'really high' hematocrit is uncommon in OSA. Systematic reviews and meta-analyses show that true secondary polycythemia occurs in only about 2% of OSA patients overall (and roughly 6% of severe OSA cases), with average hematocrit levels remaining well within normal physiological reference ranges. While OSA is considered during the differential diagnosis of unexplained secondary erythrocytosis, markedly elevated hematocrit is not a typical presentation or reliable standalone indicator of sleep apnea.

0:00:27Andy Galpinoverstatedmoderate

Having high-quality skeletal muscle makes it significantly harder to develop metabolic disorders.

"If you have high-quality muscle, it's really hard to have metabolic problems." (said at 0:00:27)

Skeletal muscle is the primary site of insulin-stimulated glucose disposal, and higher muscle quality—characterized by lower inter- and intramuscular fat infiltration (myosteatosis) and higher functional strength per unit of mass—is consistently associated with improved insulin sensitivity and a significantly lower risk of metabolic syndrome and incident type 2 diabetes. Systematic reviews and longitudinal cohorts demonstrate that poor muscle quality (such as higher low-density muscle area and intramuscular lipid accumulation) independently increases the odds and hazard of insulin resistance, metabolic syndrome, and type 2 diabetes by twofold or more. However, phrasing this relationship as making metabolic disorders 'really hard to have' overstates the absolute protective effect, as metabolic diseases are multifactorial and can still develop via hepatic insulin resistance, pancreatic beta-cell dysfunction, genetics, and dietary excess regardless of muscle quality.

0:44:14Andy Galpinoverstatedlow

Specific structures in the brain physically shrink when a person avoids pushing through discomfort and grow larger when they regularly engage in challenging tasks.

"there's specific areas of the brain that are developed when you push through on discomfort. And so there's an actual physical structure that gets smaller when you don't do that very often. It gets larger when you do it more often." (said at 0:44:14)

The speaker is referring to research on the anterior midcingulate cortex (aMCC), a brain region implicated in tenacity, effort-based cost-benefit computation, and the subjective 'will to persevere.' Cross-sectional neuroimaging studies have observed larger or preserved aMCC volume and cortical thickness in groups exhibiting sustained high effort or cognitive maintenance (such as endurance athletes and elderly 'superagers') compared to populations with reduced physical or cognitive engagement. However, claiming as a demonstrated fact that the structure dynamically physically shrinks from avoiding discomfort and grows larger from repeatedly pushing through it overstates the evidence: these observations stem primarily from cross-sectional associations, case reports of direct electrical stimulation, and narrative reviews, rather than prospective longitudinal trials proving bidirectional structural hypertrophy and atrophy in response to discomfort.

1:14:04Andy Galpinoverstatedmoderate

Having high-quality skeletal muscle mass makes the occurrence of type 2 diabetes extraordinarily rare.

"And now even this week, there's other data that got published on type 2 diabetes and muscle mass, effectively showing if you have high-quality muscle mass, it's extraordinarily rare to have type 2 diabetes." (said at 1:14:04)

Higher muscle quality (assessed by functional metrics like handgrip strength per unit of muscle mass, or by lower intramuscular fat infiltration) is associated with a significantly reduced risk of developing type 2 diabetes mellitus (T2DM). For example, a prospective cohort study of 355,209 UK Biobank participants found that individuals in the highest quintile of the muscle quality index had a 64% lower risk of incident T2DM compared to the lowest quintile (HR 0.36, 95% CI 0.34–0.39). However, describing T2DM as 'extraordinarily rare' in individuals with high muscle quality exaggerates the protection, as T2DM is multifactorial and can still develop due to beta-cell dysfunction, visceral adiposity, genetics, and age.

1:21:12Andy Galpinoverstatedmoderate

The greatest amount of AST (aspartate aminotransferase) in the human body is found in skeletal muscle, which frequently leads to elevated serum AST levels in individuals with high muscle mass.

"AST, the most amount of AST in your body is in muscle. So you will always see almost always see elevated AST in people with a lot of muscle mass." (said at 1:21:12)

While skeletal muscle contains a large reservoir of aspartate aminotransferase (AST) due to overall muscle volume, elevated serum AST in muscular individuals (such as weightlifters or bodybuilders) is primarily driven by exercise-induced muscle microtrauma and enzyme leakage rather than baseline muscle mass alone. In healthy individuals, resting AST levels generally remain within standard reference intervals unless strenuous or unaccustomed muscular exertion has occurred within the preceding days.

1:12:23Andy Galpinoverstatedlow

Elevated serum albumin levels (hyperalbuminemia) are almost exclusively caused by acute dehydration and hemoconcentration.

"it's super high in only one case basically: in the case of acute dehydration, right? Because it's a concentration issue, right?" (said at 1:12:23)

While dehydration and hemoconcentration are widely recognized as primary causes of elevated serum albumin concentrations, asserting that hyperalbuminemia occurs "in only one case basically" is overstated. Published clinical literature demonstrates that hyperalbuminemia is frequently observed in clinical settings outside of acute dehydration, including iatrogenic elevation following therapeutic albumin administration in acute and intensive care settings, as well as in stable outpatients associated with metabolic disorders, high BMI, and chronic sympathetic nervous system activation.

1:38:36Andy Galpinoverstatedmoderate

Springbok imaging software uses a 20- to 30-minute full-body MRI scan to create a three-dimensional model and individual volume analysis of every muscle in the body.

"Springbok does the same thing: 20- to 30-minute full MRI, full-body MRI scan, and it gives us a three-dimensional image of every muscle on your body." (said at 1:38:36)

Springbok Analytics uses artificial intelligence to perform rapid three-dimensional segmentation and volumetric analysis of musculature from MRI scans. However, claiming that it provides individual analysis of 'every muscle on your body' is an overstatement. The human body contains over 600 skeletal muscles; published evaluations of whole-body AI muscle segmentation algorithms (including Springbok's pipeline) quantify up to 71 individual muscles across the upper limbs, trunk, and lower limbs, rather than every anatomical muscle.

3

Needs context

0:27:18Andy Galpinneeds contexthigh

Medical residency work hours in the United States were historically reduced from up to 120 hours per week to an 80-hour weekly cap.

"More recently, just a couple years ago, they reduced the residency hours from 120 week to 80 or something like that." (said at 0:27:18)

The core assertion regarding the reduction of resident duty hours to an 80-hour cap is historically accurate, but the timeline stated is incorrect. In July 2003, the Accreditation Council for Graduate Medical Education (ACGME) instituted nationwide duty-hour limits capping the workweek at an average of 80 hours. Prior to this mandate, residents in demanding surgical and medical specialties routinely reported working 100 to 120 or more hours per week. However, this national policy was implemented in 2003 (with additional sub-rules added in 2011), rather than 'just a couple years ago'.

0:37:28Andy Galpinneeds contextlow

Orthosomnia is a clinical sleep disorder characterized by sleep obsession induced by the use of sleep trackers.

"Are you familiar with orthosomnia?... Obsessing about your sleep. These are clinical sleep disorders that are induced from sleep trackers." (said at 0:37:28)

The term "orthosomnia" was introduced by sleep researchers in 2017 to describe a phenomenon in which individuals develop an obsessive preoccupation with perfecting sleep data provided by wearable sleep trackers, which can paradoxically exacerbate sleep anxiety and self-reported sleep disturbances (Baron et al., 2017). Sleep clinicians acknowledge consumer sleep technology as a potential contributor to orthosomnia and misperceptions about sleep quality (Addison et al., 2023). However, orthosomnia is a descriptive term coined in the literature rather than a formally recognized, diagnostic clinical sleep disorder in standard classification manuals such as the International Classification of Sleep Disorders (ICSD-3) or the Diagnostic and Statistical Manual of Mental Disorders (DSM-5).

0:35:54Andy Galpinneeds contexthigh

Full FDA-cleared clinical sleep studies can be performed using a fingertip monitoring device.

"We can run again full FDA clinical sleep studies on your fingertip. It's not a tracker. It's not like a check your sleep score with proprietary algorithms, all that stuff." (said at 0:35:54)

FDA-cleared diagnostic home sleep apnea tests (HSATs) utilizing fingertip probes—specifically peripheral arterial tonometry (PAT) technology such as WatchPAT—are clinically validated medical devices used to diagnose obstructive sleep apnea, distinguishing them from non-medical consumer sleep trackers. However, referring to these devices as performing a "full" clinical sleep study requires qualification: full gold-standard clinical polysomnography (PSG) involves multi-channel laboratory monitoring including electroencephalography (EEG), electrooculography (EOG), electromyography (EMG), electrocardiogram (ECG), airflow, and respiratory effort. Fingertip PAT devices provide home sleep apnea testing by detecting autonomic vascular and oximetry changes rather than direct brainwave polysomnography.

12

Supported by research

0:08:27Andy Galpinsupportedmoderate

Between 70% and 80% of sleep disorders go undiagnosed.

"Somewhere, depending on the data source you look at, 70 to 80% of sleep disorders will go undiagnosed." (said at 0:08:27)

Epidemiological studies and clinical reviews consistently estimate that approximately 80% (often reported as 70% to 90%, depending on the specific condition and demographic dataset) of sleep disorders, particularly obstructive sleep apnea (OSA), remain undiagnosed in the general population.

0:05:31Andy Galpinsupportedhigh

High altitude significantly exacerbates sleep apnea and sleep-disordered breathing.

"And then when he went to his house in Vail, he went from moderate to extreme sleep apnea. He had no idea... That will exacerbate sleep disorders massively." (said at 0:05:31)

Exposure to moderate-to-high altitude markedly exacerbates sleep-disordered breathing and sleep apnea severity. In a randomized crossover trial evaluating patients with obstructive sleep apnea traveling from low altitude (490 m) to mountain elevations (1,860 m and 2,590 m, comparable to mountain towns like Vail at ~2,500 m), median apnea-hypopnea index (AHI) nearly doubled, rising from 47.5 events/h at baseline to 90.0-90.9 events/h at 2,590 m. This exacerbation is largely driven by hypoxia-induced respiratory instability leading to frequent central apneas and periodic breathing layered over pre-existing obstructive events.

0:18:20Andy Galpinsupportedmoderate

CPAP therapy in patients with sleep apnea lowers elevated hematocrit and hemoglobin levels.

"There's a lot of data on this. If you take somebody that has apnea and put them on CPAP, hematocrit and hemoglobin will come down." (said at 0:18:20)

Published clinical evidence supports the claim that continuous positive airway pressure (CPAP) therapy reduces hemoglobin and hematocrit levels in patients with obstructive sleep apnea (OSA). A 2022 systematic review and meta-analysis of ten trials found that CPAP therapy led to statistically significant reductions in both hemoglobin (mean reduction of 3.76 g/L) and hematocrit (mean reduction of 1.1%). Although the magnitude of reduction is modest and the underlying trials were predominantly single-arm before-and-after studies rather than large randomized placebo-controlled trials, the direction and presence of the effect are consistent across the literature.

0:18:55Andy Galpinsupportedmoderate

Low iron or ferritin levels occur in more than 30% of physically active individuals.

"And low iron does lead to sleep disruption... And that's going to happen in over probably 30% of physically active people, which people don't realize and doctors usually don't check." (said at 0:18:55)

Published literature confirms that low iron or ferritin levels affect a large proportion of physically active individuals and athletes. A systematic review and meta-analysis of 17,519 athletes across 122 studies found that 53.9% exhibited hypoferritinemia (ferritin <50 mcg/L), and 23.0% had absolute iron deficiency (ferritin <20 mcg/L). When using athletic thresholds for low iron stores (ferritin <30–50 mcg/L) or examining female athletic cohorts specifically, the prevalence routinely exceeds 30%.

0:20:15Andy Galpinsupportedmoderate

Typical sleep onset latency is between 5 and 20 minutes, and falling asleep in under 2 minutes is an indicator of sleep deprivation.

"if you look at something like sleep onset latency, how long it takes you to fall asleep at night, right? The general numbers there are going to be 5 to 20 minutes, right? Okay. So if you're falling asleep within like two minutes, that's probably a good sign that you're sleep-deprived." (said at 0:20:15)

Sleep medicine literature generally considers a sleep onset latency of 10 to 20 minutes (or roughly 5 to 20 minutes) to be typical in healthy, non-sleep-deprived individuals. In objective testing (such as polysomnography or the Multiple Sleep Latency Test), healthy controls typically demonstrate average sleep onset latencies around 10 to 15 minutes, whereas latencies under 5 minutes—and particularly under 2 minutes—reflect severe daytime sleepiness characteristic of significant sleep deprivation (such as behaviourally induced insufficient sleep syndrome) or central disorders of hypersomnolence.

0:18:57Andy Galpinsupportedmoderate

Low iron or ferritin levels cause sleep disruption.

"And low iron does lead to sleep disruption." (said at 0:18:57)

The statement is supported by clinical and observational evidence. Iron deficiency and depleted iron stores (low serum ferritin) are established drivers of sleep disruption, primarily through central nervous system dopamine dysregulation, restless legs syndrome (RLS), periodic limb movement disorder, and restless sleep disorder. Studies and reviews show that low ferritin and iron deficiency anemia correlate with prolonged sleep-onset latency, lower sleep efficiency, reduced slow-wave sleep, and greater sleep fragmentation in both children and adults, and iron supplementation frequently improves these sleep disturbances.

1:00:14Andy Galpinsupportedhigh

At 10,000 feet of elevation, there is a notable physiological effect on the body.

"Okay, that's enough to get a physiological effect, though." (said at 1:00:14)

Exposure to elevations of approximately 10,000 feet (~3,000 meters) triggers well-documented acute and chronic physiological effects caused by hypobaric hypoxia. These include decreased arterial oxygen saturation (SpO2), hyperventilation (hypoxic ventilatory response), increased resting and submaximal heart rates, reduced maximal oxygen uptake (VO2max, typically by 15% to 20%), increased pulmonary artery systolic pressure, and, over sustained exposure, erythropoietic acclimatization.

1:09:25Andy Galpinsupportedmoderate

Lifting weights through a full range of motion provides sufficient stimulus to maintain flexibility and mobility without dedicated stretching routines.

"If you're lifting weights in a full range of motion, then flexibility is in large part taken care of." (said at 1:09:25)

Multiple systematic reviews and meta-analyses of randomized and controlled trials demonstrate that resistance training (particularly through a full range of motion with external loads) produces improvements in flexibility and joint range of motion comparable to dedicated stretch-training protocols. A 2023 meta-analysis of 55 studies found no significant difference in range-of-motion gains between resistance training and stretching alone, or between resistance training combined with stretching versus stretching alone. While specialized athletic flexibility demands (e.g., gymnastics, dance) may still require dedicated stretching, general flexibility and mobility are effectively developed and maintained through loaded full-range resistance training.

1:09:35Andy Galpinsupportedmoderate

Physical activity and exercise significantly reduce the risk and assist in the prevention of dementia and Alzheimer's disease.

"And there's ample research now on the brain. So you're talking about dementia and Alzheimer's prevention heavily positively benefited by these things." (said at 1:09:35)

Extensive observational research and large meta-analyses demonstrate that regular physical activity and exercise are associated with a significant reduction in the risk of incident all-cause dementia and Alzheimer's disease (typically a 20% to 25% relative risk reduction). Evidence from systematic reviews of prospective cohorts indicates that higher overall and leisure-time physical activity levels consistently correlate with lower risks of cognitive decline and neurodegenerative disorders.

1:14:34Mark Hyman (host)supportedlow

People with type 2 diabetes have mitochondria that function roughly half as effectively as those in people without type 2 diabetes.

"When you look at type 2 diabetics, their mitochondria are functioning half as effectively as people who don't have type 2 diabetes, and that is related to exercise." (said at 1:14:34)

Cross-sectional skeletal muscle biopsy studies show that individuals with type 2 diabetes have approximately a 40% to 50% reduction in mitochondrial respiratory chain enzyme activity (such as NADH:O2 oxidoreductase and subsarcolemmal electron transport chain activity) compared to non-diabetic controls. This functional deficit is closely linked to lower mitochondrial density and physical inactivity, and exercise training interventions have been shown to substantially restore mitochondrial volume and oxidative capacity. Because the primary human data rely on small cross-sectional biopsy cohorts (n = 10–12 per group), certainty is low.

1:06:06Andy Galpinsupportedmoderate

Serum magnesium is a poor indicator of total body magnesium status because the majority of magnesium in the human body is stored in bone.

"So not just serum magnesium—that's a really bad way to understand magnesium in the system, right, because it's almost all in bone—but something like magnesium RBC" (said at 1:06:06)

Published reviews confirm that serum magnesium is a poor indicator of total body magnesium status. Less than 1% to 2% of total body magnesium is located in blood and extracellular fluid, with the vast majority sequestered in bone and intracellular tissues. Because serum magnesium levels are homeostatically defended through bone exchange and renal conservation, normal serum levels can persist despite significant tissue depletion. Erythrocyte (RBC) magnesium measures intracellular concentration and provides a better indicator of cellular magnesium exposure over time.

1:36:00Mark Hyman (host)supportedhigh

The cost of sequencing a whole human genome has decreased from approximately one billion dollars to around $300.

"And so what was a billion-dollar test to get your genome sequenced is now $300." (said at 1:36:00)

The cost of sequencing a human genome has dropped dramatically over the past two decades. The initial sequencing of the first human genome via the international Human Genome Project took over a decade and cost roughly $2.7 to $3 billion (with Celera's parallel shotgun sequencing effort costing approximately $100 million). Following the advent and scaling of next-generation sequencing (NGS) and high-throughput platforms, the cost per human whole genome dropped to approximately $1,000 in the mid-2010s and has further declined to around $200–$300 on high-throughput modern platforms.

6

No source found (not proven false)

0:08:27Andy Galpinunverifiedvery low

Sleep regularity (going to bed and waking up at the same time every day) has a larger impact on perceived daytime energy levels than total sleep duration.

"Sleep regularity is more important than people think, meaning going to bed and waking up at the same time every day. When it comes to perceived feeling, your energy levels, that regularity will play a bigger impact than the total hours will." (said at 0:08:27)

No published record matching the claim that sleep regularity has a larger impact on perceived daytime energy levels than total sleep duration was located; this does not prove the claim false.

0:30:28Andy Galpinunverifiedvery low

An overnight respiratory rate of 17 breaths per minute is elevated, especially in cardiovascularly fit individuals.

"You have overnight respiratory rate of 17 breaths per minute, which is high. This is not somebody who's cardiovascularly unfit." (said at 0:30:28)

No published record matching the claim that an overnight respiratory rate of 17 breaths per minute is abnormally elevated or specifically indicative of pathology in cardiovascularly fit individuals was located; this does not prove the claim false.

1:05:22Andy Galpinunverifiedvery low

The traditional formula for maximum heart rate (220 minus age) was not derived from rigorous scientific trials, but was originally approximated informally on an airplane ride for a conference presentation.

"So your maximum heart rate, that number 220 minus your age, the story, the lore is that was actually written out on like the back of a sheet on a plane ride. So a couple of guys had to do a conference and they had to have some way to kind of present something, and they're just like, "Uh, 220," ran some numbers, like, "Yeah, that works out pretty well."" (said at 1:05:22)

No published record matching the claim that the traditional maximum heart rate formula (220 minus age) was originally approximated informally on an airplane ride for a conference presentation was located; this does not prove the claim false.

1:18:24Andy Galpinunverifiedvery low

Overtraining and excessive physiological stress lead to marked reductions in circulating DHEA, total testosterone, and free testosterone.

"Stress response goes up when you're training too much. And every single time in that exact example, what do you think happens to DHEA pools? What do you think happens to total testosterone? What happens to free testosterone? HOST: They're plummeted, right? GUEST1: Yeah." (said at 1:18:24)

No published record matching the claim that overtraining and excessive physiological stress lead to marked reductions in circulating DHEA, total testosterone, and free testosterone was located; this does not prove the claim false.

1:34:43Andy Galpinunverifiedvery low

Professional golfer Jon Rahm stopped using wearable sleep trackers like Whoop or Oura because the sleep tracking data was causing negative psychological issues.

"Some of our athletes, Jon Rahm, professional golfer, he's said this a bunch publicly, so I can say it. The Whoop or the Oura had to get taken off. The sleep tracking was causing issues with him." (said at 1:34:43)

No published record matching the claim that professional golfer Jon Rahm stopped using wearable sleep tracking devices (such as Whoop or Oura) due to psychological issues or anxiety stemming from sleep tracking data was located; this does not prove the claim false.

1:43:14Mark Hyman (host)unverifiedvery low

Detecting cancer at stage I or stage II yields a 5-year relative survival rate exceeding 95%.

"when you pick it up in stage I or II, it's over 95% survival at 5 years, which is, you know, basically a cure." (said at 1:43:14)

No published record matching the claim that detecting cancer at stage I or stage II yields a 5-year relative survival rate exceeding 95% was located; this does not prove the claim false.

Unverified means no publication matching the claim was located; it does not prove the claim false. Spotted an error? See the corrections policy - disputes from the people quoted are prioritized.