HoldsUp

Dr. Ford Brewer MD MPH · 2026-07-02 · Ford Brewer (host), Jesus Vega, Tom, Mark, James, Scape, Emily, Quinn, Stan, George

Don't Eat Junk Your Body Can't Handle

31 research-tied claims examined: 4 contradicted 2 overstated 4 context 16 supported 5 unverified

4

Contradicted by research

0:14:04Ford Brewer (host)contradictedhigh

A measurement of 1.3 mm or higher on a carotid intima-media thickness (CIMT) test is classified as discrete plaque.

"In the bottom half, it starts with a thing which says something like uh plaque analysis and it says if you have 1.3 or higher then that then that's classified as what I would call discrete plaque." (said at 0:14:04)

Standard international consensus guidelines do not define discrete carotid plaque as a thickness of 1.3 mm or higher. According to the widely adopted Mannheim Carotid Intima-Media Thickness and Plaque Consensus, carotid plaque is defined as a focal wall encroachment into the arterial lumen of at least 0.5 mm, 50% or more of the surrounding IMT value, or an absolute total thickness of greater than 1.5 mm. While some proprietary commercial test reports or software may utilize non-standard arbitrary thresholds, established clinical consensus defines the absolute thickness threshold for plaque at >1.5 mm rather than 1.3 mm.

0:50:48Ford Brewer (host)contradictedhigh

Grinding whole wheat berries at home renders the flour ultra-processed.

"there's some potential that getting your own wheat berries helps, but usually when you grind it up, you're again, your grinding it up is making it ultra-processed." (said at 0:50:48)

Under the internationally recognized NOVA food classification system, physical and mechanical operations applied to whole foods—such as grinding, milling, crushing, or drying whole grains—produce minimally processed foods (Group 1). Ultra-processed foods (Group 4) are defined by industrial formulations containing additives (such as emulsifiers, preservatives, or artificial flavorings) and industrial ingredients derived from foods through processing techniques not typically performed at home. Grinding whole wheat berries at home results in simple whole-wheat flour without additives or chemical extraction, meaning mechanical grinding does not make the food ultra-processed.

0:55:36Ford Brewer (host)contradictedmoderate

Half of all heart attacks result in sudden death.

"With heart attack, half of them result in sudden death. You wake up dead." (said at 0:55:36)

The speaker conflates several cardiovascular epidemiology statistics. It is not true that 50% of all heart attacks result in sudden death. Rather, published epidemiological data (such as from the Nurses' Health Study and Framingham Heart Study cohorts) demonstrate that sudden cardiac death (SCD) accounts for approximately half of all cardiac *deaths*, and that SCD frequently occurs as the initial clinical manifestation of undiagnosed coronary artery disease.

1:17:48Ford Brewer (host)contradictedhigh

A coronary artery calcium score of 1,000 or above often indicates plaque stabilization and calcification associated with lower risk than lower scores with unstable plaque.

"having a calcium score 1,000 or above is often a marker that you've gotten your metabolic disease in line and that you have less risk than people with lower calcium scores... due to the stabilization, i.e., that plaque burden went up because the soft plaque is now hard plaque or calcified." (said at 1:17:48)

The claim that a coronary artery calcium (CAC) score of 1,000 or greater indicates lower cardiovascular risk than lower scores due to plaque stabilization is contradicted by clinical evidence. Large prospective cohort data, such as from the Multi-Ethnic Study of Atherosclerosis (MESA), demonstrate that individuals with CAC ≥ 1,000 are at very high cardiovascular risk (a 4.71-fold higher risk for all CVD events vs CAC 0 and a 1.65-fold higher risk vs CAC 400-999), with event rates comparable to patients with established secondary prevention disease. While increased calcium density at a given volume level is associated with plaque stabilization, an overall score of ≥ 1,000 reflects massive total coronary plaque burden and unequivocally denotes high risk rather than lower risk or controlled metabolic disease.

2

Overstated

1:04:22Ford Brewer (host)overstatedmoderate

Metabolic disease affects over half of the population starting at age 18 and worsens every year thereafter.

"since metabolic disease is such a problem for over half of us starting at age 5 at 18 and then getting worse every year after that." (said at 1:04:22)

The claim that metabolic disease affects over half of the population starting at age 18 is an overstatement. Nationally representative cross-sectional data from the United States (NHANES) show that the overall prevalence of metabolic syndrome in adults is approximately 34% to 35%, and it is significantly lower in young adults (aged 20-39 years, where prevalence is around 15-20%) rather than exceeding 50% at age 18. While metabolic syndrome prevalence does progressively increase with age (exceeding 45-50% in older adults aged 60 and older), and broadly defined suboptimal cardiometabolic health (having at least one suboptimal marker) is common across adulthood, metabolic disease/syndrome itself does not affect over half the population starting at age 18.

1:19:22Ford Brewer (host)overstatedmoderate

Postprandial blood glucose spikes above 140 mg/dL cause arterial damage.

"If your body's making that sugar, then it's usually not going to make it to an extent where it burns those arteries. You know, where you want to start worrying about it is going over 140 and being associated with eating foods." (said at 1:19:22)

Postprandial hyperglycemia exceeding 140 mg/dL (7.8 mmol/L, the clinical cutoff for impaired glucose tolerance) is an established independent cardiovascular risk factor and can acutely trigger transient endothelial dysfunction, oxidative stress, and vascular inflammation in experimental and clinical settings. However, describing post-meal blood glucose spikes above 140 mg/dL as 'burning' arteries overstates the biology: transient postprandial elevations induce reversible impairments in flow-mediated dilation rather than acute arterial destruction, and brief excursions around or above 140 mg/dL can occur in healthy individuals following carbohydrate ingestion without causing immediate vascular damage.

4

Needs context

0:01:41Ford Brewer (host)needs contextmoderate

Approximately 80% of Americans eat cereal.

"Now, most of the people that are watching our videos uh really are not eating cereal, but when you think about it maybe or look it up, 80% of Americans are eating cereal." (said at 0:01:41)

The claim that approximately 80% of Americans eat cereal is roughly consistent with US consumer market research and household penetration surveys (which commonly report that 80% to 86% of Americans or US households purchase and consume cold breakfast cereal). However, based on nationally representative dietary survey data from the National Health and Nutrition Examination Survey (NHANES), on any given single day only about 19% of US adults report consuming ready-to-eat cereal. Thus, the 80% figure reflects general consumer/household penetration rather than daily consumption.

0:27:47Jesus Veganeeds contextmoderate

The Somogyi effect occurs when blood sugar drops very low in the early morning, triggering reactive fasting hyperglycemia.

"And there's also something very interesting a lot of people don't know, which is called the Somogyi effect, which is similar to the dawn effect, but this effect happens when your blood sugar goes extra low, very low in the early morning, and that's a trigger to high blood sugar, fasting blood sugar." (said at 0:27:47)

The speaker accurately defines the classical clinical concept of the Somogyi effect (or Somogyi phenomenon): rebound fasting hyperglycemia hypothesized to occur in response to nocturnal or early-morning hypoglycemia via counter-regulatory hormone release. However, modern clinical research using continuous glucose monitoring has largely challenged this hypothesis, demonstrating that nocturnal hypoglycemia rarely causes rebound morning hyperglycemia (morning glucose levels are typically lower after nocturnal hypoglycemic episodes, not higher) and that morning hyperglycemia is more often driven by the dawn phenomenon or waning overnight insulin levels.

1:02:59Jesus Veganeeds contextmoderate

The most effective intervention to resolve sleep apnea is weight loss.

"the best thing you could do to fix a sleep apnea is to lose weight." (said at 1:02:59)

Weight loss is a highly effective, foundational lifestyle intervention for individuals with obstructive sleep apnea (OSA) who have overweight or obesity, significantly reducing the Apnea-Hypopnea Index (AHI) and associated cardiometabolic risks. However, calling it unconditionally the "best thing to fix" sleep apnea requires context: continuous positive airway pressure (CPAP) remains the clinical gold standard and first-line treatment for immediately eliminating airway obstruction, and weight loss alone often substantially reduces rather than completely resolves moderate-to-severe OSA. Furthermore, OSA in individuals without excess body weight may be driven primarily by craniofacial or other non-weight-related anatomical factors.

1:15:55Ford Brewer (host)needs contextmoderate

Carotid Intima-Media Thickness (CIMT) ultrasound can directly detect and visualize soft arterial plaque.

"One of the next places I will go is how about an old-fashioned CIMT? That's going to show me soft plaque." (said at 1:15:55)

Carotid duplex and B-mode ultrasound can identify carotid atherosclerotic plaques and evaluate their acoustic characteristics—specifically identifying echolucent (hypoechoic) areas, which are often described colloquially as 'soft plaque' and correlate with lipid-rich necrotic cores or intraplaque hemorrhage. However, the claim conflates Carotid Intima-Media Thickness (CIMT) measurement with plaque imaging/characterization. By standard consensus definitions (such as the Mannheim consensus), CIMT specifically measures the thickness of the intima-media layer in plaque-free arterial wall segments to assess early vascular remodeling, whereas distinct focal protrusions are classified as plaques. Furthermore, while quantitative ultrasound can image echolucent plaques, its diagnostic accuracy for reliably characterizing specific vulnerable components (like lipid-rich necrotic cores versus intraplaque hemorrhage) remains variable compared to advanced modalities such as high-resolution MRI.

16

Supported by research

0:05:30Ford Brewer (host)supportedmoderate

Repeated glucose and insulin spikes wear down and damage the endothelial glycocalyx lining of artery walls, initiating plaque formation.

"Do that a few times a day, every day for a few years, and it wears down that inner lining of your artery wall, the glycocalyx. That's part of the endothelium. Think of it as that smooth inner surface of the pipe that your blood flows through. Those repeated spikes irritate it. They inflame it. They even damage it. When that inflamed, damaged wall is exactly where plaque starts to build." (said at 0:05:30)

The endothelial glycocalyx is a carbohydrate-rich, gel-like protective layer of proteoglycans and glycoproteins lining the luminal surface of vascular endothelial cells. Clinical physiological studies demonstrate that acute hyperglycemia causes rapid degradation and shedding of the glycocalyx layer via oxidative stress mechanisms. In human trials, acute experimental hyperglycemia halved systemic endothelial glycocalyx volume and led to endothelial dysfunction and coagulation activation. Destruction of the glycocalyx increases vascular permeability, facilitates low-density lipoprotein (LDL) infiltration, and permits leukocyte adhesion to the underlying endothelium, representing early initiating steps in atherosclerotic plaque formation.

0:24:35Ford Brewer (host)supportedmoderate

Cortisol or epinephrine release upon waking or exercising fasted causes the liver to release stored glucose into the bloodstream.

"When you wake up or when you have a cortisol effect, that cortisol or epinephrine, adrenaline that you get from walking or just from waking up and f after your fast. Both of those can cause you to release cortisol or epinephrine and those in turn can cause your liver to release blood sugar." (said at 0:24:35)

Physiological studies confirm that early morning waking and exercise during fasting stimulate counterregulatory hormones, including cortisol and epinephrine (adrenaline), which promote hepatic glucose output (via glycogenolysis and gluconeogenesis) into the bloodstream.

0:25:28Ford Brewer (host)supportedmoderate

The HbA1c test misses over half of insulin resistance or metabolic health problems.

"No, not an A1C. The A1C misses over half of this problem. I'm talking about where you get fasting glucose and fasting insulin, then you take the sugar, an hour later you get glucose and insulin, and then an hour after that you get glucose and insulin." (said at 0:25:28)

Large population-based studies confirm that standard HbA1c thresholds miss more than half of cases of dysglycemia detected by oral glucose tolerance tests (OGTT). In national cohort evaluations (such as NHANES data), standard HbA1c screening cutoffs had sensitivities of only ~25% to 35% for detecting diabetes and prediabetes compared with OGTT-based classifications, yielding false-negative rates exceeding 60% to 75%. Furthermore, HbA1c reflects average blood glucose rather than dynamic insulin secretion; individuals with early insulin resistance frequently maintain normal glycemic and HbA1c levels through compensatory hyperinsulinemia, which is only captured when dynamic glucose and insulin responses are measured.

0:31:55Ford Brewer (host)supportedmoderate

Approximately 90 percent of metabolic disease cases have not been diagnosed by typical healthcare providers.

"And Tom, it sounds like you know already 90% of this has not been diagnosed by by your doc, by the typical doc out there and the typical health care provider." (said at 0:31:55)

Nationally representative epidemiological data from the National Health and Nutrition Examination Survey (NHANES) consistently show that approximately 85% to 92% of adults with prediabetes—a primary marker of early metabolic disease—are unaware of their condition and have not received a formal diagnosis from a healthcare provider. For example, an analysis of NHANES data found that 92.0% of U.S. adults with prediabetes were unaware of their condition (PMID: 27612580), and an earlier NHANES analysis noted that only 4.8% of individuals meeting prediabetes criteria reported having received a formal diagnosis from their physician (PMID: 20724649).

0:34:24Ford Brewer (host)supportedmoderate

Surveys from institutions such as Johns Hopkins found that three-quarters of primary care doctors and cardiologists do not know how to diagnose metabolic disease or prediabetes.

"multiple surveys have been done from places like Hopkins, and what they found was three-quarters of the local doctor, your local doctor who the current administration is referring you back to, don't know how to diagnose this problem. Three-quarters of them—internists, family practitioners, cardiologists—don't know how to diagnose this problem." (said at 0:34:24)

Surveys led by researchers at Johns Hopkins Bloomberg School of Public Health and Johns Hopkins School of Medicine examined primary care providers' (PCPs') knowledge of prediabetes screening, diagnostic criteria, and management. In a survey of mid-Atlantic primary care providers (Tseng et al., 2017), only 17% of PCPs correctly identified the laboratory parameters for diagnosing prediabetes using both fasting glucose and HbA1c (meaning 83%, or over three-quarters, failed to identify the full diagnostic criteria). A subsequent national survey of US PCPs (Tseng et al., 2019) similarly demonstrated widespread knowledge gaps regarding prediabetes laboratory diagnostic criteria and screening guidelines.

0:36:40Jesus Vegasupportedmoderate

Repeated, continuous spikes of glucose and insulin cause arterial plaque formation.

"it's the continuous glucose spikes and insulin spikes what's causing the problem—the over and over and overexposure to high glucose and high insulin causing arterial plaque." (said at 0:36:40)

Substantial observational and mechanistic evidence confirms that repeated acute glucose spikes (glycemic variability) and elevated insulin levels (hyperinsulinemia) contribute directly to endothelial dysfunction, vascular inflammation, and the initiation and progression of arterial plaques. Continuous glucose monitoring and intravascular imaging studies show that acute glucose excursions provoke oxidative stress and correlate significantly with arterial endothelial damage, plaque instability, and coronary plaque vulnerability.

0:37:18Jesus Vegasupportedmoderate

Patients with diabetes typically experience a 5- to 7-year delay before receiving a formal diagnosis.

"To think about that for somebody who has diabetes you have to wait for five to seven years to get the diagnosis done, it's—I learned this word three weeks ago: preposterous." (said at 0:37:18)

Epidemiological models evaluating the prevalence and progression of diabetic retinopathy at the time of clinical diagnosis estimate that the asymptomatic onset of type 2 diabetes typically precedes formal clinical diagnosis by approximately 4 to 7 years (with refined models estimating an average delay of about 6 years).

0:55:28Ford Brewer (host)supportedmoderate

Half of all heart attacks occur in people with normal cholesterol levels.

"Half of all heart attacks happen in people with perfectly normal cholesterol." (said at 0:55:28)

A large observational study of 136,905 hospitalizations for coronary artery disease (including acute myocardial infarction) across 541 US hospitals found that almost half of patients admitted with CAD had low-density lipoprotein (LDL) cholesterol levels under 100 mg/dL, which was considered the standard optimal/normal threshold (Sachdeva et al., 2009). The mean LDL cholesterol level at admission was 104.9 mg/dL. While the majority of these patients had abnormal total lipid profiles when considering low high-density lipoprotein (HDL) cholesterol or high triglycerides, the assertion that roughly half of acute coronary events occur in individuals with normal or optimal LDL cholesterol levels is well-supported by observational registry data.

0:59:40Ford Brewer (host)supportedhigh

Beetroot and beetroot powder increase nitric oxide levels in the body.

"beetroot, a lot of people say, you know what, beets are great. They can help you actually increase your NO, nitric oxide." (said at 0:59:40)

Beetroot and beetroot products are rich in inorganic dietary nitrate (NO3-). Following ingestion, dietary nitrate is reduced to nitrite (NO2-) by commensal oral bacteria, which is then absorbed and further reduced to nitric oxide (NO) in the bloodstream and tissues through the enterosalivary nitrate-nitrite-NO pathway. Extensive randomized controlled trials and systematic reviews demonstrate that beetroot supplementation significantly elevates plasma nitrate and nitrite concentrations (the primary circulating biomarkers of nitric oxide production) and enhances nitric oxide bioavailability, leading to measurable physiological effects such as reduced blood pressure and improved vascular function.

1:00:25Ford Brewer (host)supportedhigh

Creatine supplementation converts to creatinine and can falsely suggest kidney failure on standard blood tests, whereas cystatin C testing avoids this artifact.

"creatine gets turned into creatinine and creatinine is how doctors measure the health of your kidneys. So sometimes people will take creatine, they increase their creatinine and their doctor thinks they're having kidney failure and that's not what's going on... And one of those is a cystatin C" (said at 1:00:25)

The speaker's claim is supported by randomized controlled clinical trial data. Creatine spontaneously degrades non-enzymatically into creatinine, which can raise serum creatinine levels and lead to a false clinical impression or misdiagnosis of renal impairment on standard creatinine-based tests. Using cystatin C (a marker independent of creatine intake and muscle mass) demonstrates normal glomerular filtration rate and confirms that renal function remains unaltered.

0:30:14Tomsupportedhigh

The USDA Dietary Guidelines recommend keeping saturated fat consumption to less than 10 percent of total daily calories.

"they're still recommending we eat four to five servings of fruits and vegetables and four servings of grains a day and keep our saturated fat to less than 10%?" (said at 0:30:14)

The claim is accurate. The USDA/HHS Dietary Guidelines for Americans consistently recommend limiting saturated fat intake to less than 10 percent of total daily calories.

1:03:28Ford Brewer (host)supportedhigh

Sleep apnea can occur in individuals with a normal BMI, including a BMI of 21.

"And you don't have to be fully overweight or obese to be dealing with sleep apnea... I had sleep apnea when my BMI was 21." (said at 1:03:28)

Obstructive sleep apnea (OSA) is not restricted to overweight or obese individuals and commonly occurs in people with a normal body mass index (BMI). An individual participant data meta-analysis of 12,860 adults across four community-based cohorts found that approximately 23.5% of adults diagnosed with OSA were normal weight or underweight (BMI < 25 kg/m²). In non-obese individuals, OSA is frequently driven by non-adipose factors, such as craniofacial skeletal morphology and airway anatomy.

1:10:42Ford Brewer (host)supportedhigh

Colchicine is an anti-inflammatory drug identified through Harvard research by Paul Ridker to reduce cardiovascular risk.

"colchicine is clearly an option. It's an it's an anti-inflammatory. And uh some very smart people uh Paul Ridker, Gavin Blake up at Harvard noticed that that statins actually are anti-inflammatories. So they went down this whole research train for the for the next 30 years looking at anti-inflammatories to see if they can decrease risk. Sure enough, that's how colchicine came up." (said at 1:10:42)

Substantial clinical trial and review evidence demonstrates that low-dose colchicine is an anti-inflammatory therapy that significantly reduces major adverse cardiovascular events (MACE) in patients with coronary artery disease and after myocardial infarction, leading to FDA approval for cardiovascular risk reduction. This development directly followed decades of research—spearheaded by Paul Ridker, Gavin Blake, and colleagues at Harvard and Brigham and Women's Hospital—establishing the inflammatory hypothesis of atherosclerosis and showing that statins and dedicated anti-inflammatory drugs confer cardiovascular protection by reducing vascular inflammation.

1:17:35Ford Brewer (host)supportedhigh

Gentle, slow walking in Zone 2 or below decreases blood sugar and blunts glucose spikes.

"just taking a gentle, slow walk. So, getting back to zone two or even less, you know, a gentle walk will often help you decrease that blood sugar and keep it down" (said at 1:17:35)

Randomized controlled trials and meta-analyses consistently demonstrate that gentle, light-intensity walking (or light activity performed at low aerobic intensities, such as Zone 2 or lower), especially when performed after a meal or to interrupt prolonged sitting, significantly lowers postprandial blood glucose levels and blunts glucose excursions compared to remaining seated or inactive.

1:14:45Ford Brewer (host)supportedhigh

Performing additional coronary procedures has been proven not to prevent heart attacks in stable patients.

"Then my question is, okay, we know that doing more procedures has been demonstrated it doesn't prevent a heart attack. What are you trying to cure at this point?" (said at 1:14:45)

Large-scale randomized controlled trials have demonstrated that in patients with stable ischemic heart disease, routine coronary revascularization (such as percutaneous coronary intervention [PCI] or coronary artery bypass grafting [CABG]) added to guideline-directed optimal medical therapy does not reduce the incidence of myocardial infarction or all-cause mortality compared to optimal medical therapy alone. The primary benefit of revascularization in this population is symptom and angina relief rather than the prevention of future heart attacks or death.

1:33:20Jesus Vegasupportedmoderate

Cardiac imaging scans can simultaneously assess both myocardial perfusion and plaque characterization to identify plaque type.

"I don't think both assessments are opposite, like you can have both the perfusion, but you can also have the plaque characterization where you know what type of plaque you have." (said at 1:33:20)

Contemporary non-invasive cardiac imaging, particularly comprehensive cardiac computed tomography (CCTA combined with CT myocardial perfusion imaging or CT-derived fractional flow reserve), enables the simultaneous evaluation of both functional myocardial perfusion/ischemia and anatomical vessel wall features. This includes plaque characterization such as identifying calcified, non-calcified, mixed, or high-risk low-attenuation plaque components in a single clinical session.

5

No source found (not proven false)

0:06:20Ford Brewer (host)unverifiedvery low

NHANES survey data shows that over half of 18-year-olds in the US have some degree of insulin resistance.

"The NHANES, National Health and Nutrition Examination Survey, which was part of that survey, the census survey, looked and did testing and it found, hey, it's not 60-year-olds that have this problem. Yes, as you get older it's more common, but even at age 18, 18 years old, over half of us already have some degree of insulin resistance, and it gets worse every year." (said at 0:06:20)

No published record matching the claim that NHANES survey data shows over half of 18-year-olds in the United States have insulin resistance was located; this does not prove the claim false.

0:06:58Ford Brewer (host)unverifiedvery low

By age 60, the majority of adults have some degree of insulin resistance.

"And most people can't handle these foods because by our 60s, most of us already have some degree of insulin resistance." (said at 0:06:58)

No published record matching the claim that the majority of adults have some degree of insulin resistance by age 60 was located; this does not prove the claim false.

0:30:14Tomunverifiedvery low

The 2025 USDA dietary guidelines state that individuals with certain chronic diseases may experience improved health outcomes when following a low carbohydrate diet.

"Here's what the guidelines 2025—I guess they came out this year. It says, "Individuals with certain chronic diseases may experience improved health outcomes when following a low carbohydrate diet. Work with your health care professional to identify and adopt a diet that is appropriate for you and your health condition."" (said at 0:30:14)

No published record matching the claim that the 2025 USDA dietary guidelines state that individuals with certain chronic diseases may experience improved health outcomes when following a low carbohydrate diet was located; this does not prove the claim false.

1:15:36Ford Brewer (host)unverifiedvery low

Infection-induced cortisol elevation typically raises blood sugar to 120-130 mg/dL for three to fifteen days.

"And I think the big issue is cortisol. Why are you having cortisol? Because you had an infection. To your point, how long does that uh should you see these 120s and 130s? And it's it's impossible to predict. It's usually not going to be a month, but it's usually anywhere from three to 15 days." (said at 1:15:36)

No published record matching the claim that infection-induced cortisol elevation typically raises blood glucose to 120-130 mg/dL for three to fifteen days was located; this does not prove the claim false. While acute infection triggers neuroendocrine stress responses—including cortisol and catecholamine release leading to stress-induced hyperglycemia—the magnitude and duration of glycemic elevation vary widely according to the severity of illness and individual metabolic factors, rather than following a defined 3- to 15-day timeframe or specific 120-130 mg/dL concentration range.

1:17:05Ford Brewer (host)unverifiedvery low

High-intensity interval training (HIIT) can raise blood glucose to 120-140 mg/dL in fat-adapted individuals through cortisol and epinephrine secretion.

"quite often when you're fat-adapted, even just some HIIT training will crank that blood sugar up to 120, 130, 140... whether it's from dawn-effect-related cortisol or from HIIT-training-related cortisol and epinephrine" (said at 1:17:05)

No published record matching the claim that high-intensity interval training (HIIT) raises blood glucose to 120-140 mg/dL in fat-adapted individuals through cortisol and epinephrine secretion was located; this does not prove the claim false.

Unverified means no publication matching the claim was located; it does not prove the claim false. Spotted an error? See the corrections policy - disputes from the people quoted are prioritized.